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The Basic Principles Of Werner syndrome RecQ helicase-IN-1

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It is intriguing to notice that if the DCM-producing mutation K36Q in cTnI was released in the existence of Ca2+, the simulation showed that Ser 22 and 23 no longer interacted intently with cTnC, in accord with our speculation the Ca2+-cTnC-cTnI N terminus interaction is exclusive which is destabilized right https://johnnyglqvy.weblogco.com/28051624/manual-article-review-is-required-for-this-article

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